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When the heterozygote genotype possesses higher relative fitness than either the homozygous dominant or homozygous recessive genotypes.
I would guess that a mutant mouse that has no, that doesn't have some of these genes for circadian rhythm, well, for them, it's just gonna be, it's just gonna be random, it's just gonna be much more sporadic.
So, much, much more sporadic, sporadic activity, activity, fewer, fewer continuous, continuous, or I would say, maybe, less continuous periods, or shorter continuous periods, shorter continuous periods of activity and inactivity, activity slash inactivity. So that was my predictions that I would see for mutant mice in either of these. In nature, mice are potential prey for some predatory birds that hunt during the day.
Mouse, then, then the mouse is more likely to be eaten, eaten if it is active during the day, if it is active during the day.
And if it is, I guess you could say that's one, that's one feature of our model, that you're just more likely to be eaten if you're active when the birds are hunting.
In animals and plants, there is only about one mutation in every 100,000 genes per generation. Define the following: Genetic drift: unpredictable fluctuations in allele frequencies, reduces genetic variation over time through such losses of alleles Bottleneck effect: when environmental change greatly reduces a population, the ratio of genes is mixed up.
There is a better chance that the disease will only affect a small portion of the population. The mutation rates rise as the generation spans decrease.
With this information one can predict how the disease will affect a population in the future. What are the two broad processes that make evolution possible? Point mutation: Has impact in phenotype, like a sickle cell disease, most however are harmless.
If you have the percentage of people with a disease and if you know weather the disease is recessive than it is easy to find the percentage of a population that are carriers of the disease.
To investigate the claim that exposure to light overrides the genetically-controlled circadian rhythm, the researchers plan to repeat the experiment with mutant mice lacking a gene that controls the circadian rhythm. So under some mutant mice, mutant mice, under, so let me make two columns, mutant mice under L12, L12: D12, and then under continuous, continuous darkness, what would I expect? They didn't get, this isn't a gene that somehow makes them not sensitive to light.
Predict the observed activity pattern of the mutant mice under L12: D12 conditions and under DD, continuous darkness conditions, that would support the claim that light overrides the genetically-controlled circadian rhythm. So I would assume under the L12: D12, they would behave, they would have activity pattern similar to non-mutant mice, activity same as non-mutant, as non-mutant, mutant mice, non, or inactivity, inactivity during L12, activity, activity during D12. They still could react to the light and the darkness the same way that a non-mutant mice would. The non-mutant mice, we said that they, they went off of a 24-hour cycle, but they still had a cycle where they were inactive, active, inactive, active, and it was less than 12 hours for each cycle, but it was close to it.